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Endogenous peptide with enzymatic properties produced in the kidney and excreted in the urine. Converts plasminogen to plasmin, causing fibrinolysis and dissolution of thrombi.
Indicated in the treatment of arterial, pulmonary and venous thrombosis.
Intravenous administration.
At the date of the last update, there was no available published data on its excretion in breast milk.
Its high molecular weight and rapid elimination (AEMPS 2016, FDA 2008) make it very unlikely to pass into breast milk in a clinically significant quantity.
Due to its protein nature, it is inactivated in the gastrointestinal tract, not being absorbed (practically zero oral bioavailability), which makes it difficult or impossible to pass into the infant’s plasma from ingested breast milk, except in premature infants and in the immediate neonatal period in infants, during which there may be increased intestinal permeability
Expert authors consider the use of this medication safe during breastfeeding (Briggs 2015, Schaefer 2015).